Management of Nerve Injuries
Following points to be included in one's scheme .
A. Detailed Clinical History
1. Particular detatils of patient
Hand dominance
Profession
2. C/C
3. History of Present illness
Regarding
Site of Injury
mechanism of injury -blast , crush , tractrion ,
open wound with contamination
weakness , progressing or improving
consequences of weakness on daily life
abilities related to the organ affected
constitutional symptoms
Any other injuries on body
4. Treatment history -
Emergency management
dressing , debridement , tag sutures any ?
Definitive Management
end -end anastomosis , end to soft tissue to prevent contraction
coverage of soft tissues
PT document use of dynamic or static splints
Active or passive motion ?
Its outcome
any contractures , infection ,
improvement in motor function
improvement in sensation
improvement in functions
5. Past history
TT prohylaxis
drug allergy
DM and other medical illnesses
6. Expectation of Patient
B. Examination - Nerve examination mentioned in previous sections
C.Investigations
1. Electrodiagnostic tests
document injuries , location of inslt , severity of injury , recovery pattern , prognosis ,
objective data for impairment documentation , pathology , selection of optimal muscles for
tendon trasnsfers
Nerve Conductions test (NCT ) and Electromyography (EMG )
shows nerve conductivity , axon or myelin involvement and muscles recruitment capability
Nerve Conduction test
immediately after injury - interference of conduction but impossible to determine severity
Proximal and distal to injury - Normal response
Stimulation across injured segment varies
5-10 days - Reduction in amplitude
- altered configuration of evoked potential
- Neuropraxia - conduction distal to lesion is normal .
Injury Pattern Porximal Stimulation Immediately after injuries after 10 days
(conduction distal to lesion) (conduction across lesion ) (across lesion)
Neuropraxia No response Normal response Normal
Axonotemesis No response Normal No response
or neurotemesis
Proximal or central neurological lesions accessed using retrograde techniques (e.g. F-waves , SSEP )
EMGs
10-14 days after neural injury
spontaneuous rest potentials + sharp waves appear in innervated myotomes with axonal injury
14 to 18 days
fibrillation potentials
denervation potential - last until innervated.
3 Weeks
neural sprouting and increase in amplitude.
2 to 6 months
large potentials remain until reinnervation completed.
Normal insertional activity present immediately after insertional activity.
Sharp waves
Amplitude - 100 - 400 microVoltage
Duration 5 - 150ms
Rate ; 2 - 40 Hz
wide body complexes
Fibrillation
Amplitude ; 5 - 1000 microvoltage
Duration ; 0.5 - 2 ms
Rate; 2-3 Hz.
narrow body
onc can check the waves ;
https://www.researchgate.net/figure/Positive-sharp-waves-and-fibrillation-potentials-were-evident-in-all-four-limbs-upon_fig2_26881420
D.Treatment
Physiotherapy - ROM of the joints to prevent contracutre of joints
Splints
Nerve Repair -we will discuss later on different type of Nerve Repair. Upto now be able to examine
them properly from previous topics .
Tendon Transfer - be able to know different donor tendons for different losses in different nerve
injuries more importantly in Upper Extremity. Radial Nerve examination and its losses is thoroughly
testested in examination.
Its equally important for median , ulnar nerves and brachial plexus injury.
A. Detailed Clinical History
1. Particular detatils of patient
Hand dominance
Profession
2. C/C
3. History of Present illness
Regarding
Site of Injury
mechanism of injury -blast , crush , tractrion ,
open wound with contamination
weakness , progressing or improving
consequences of weakness on daily life
abilities related to the organ affected
constitutional symptoms
Any other injuries on body
4. Treatment history -
Emergency management
dressing , debridement , tag sutures any ?
Definitive Management
end -end anastomosis , end to soft tissue to prevent contraction
coverage of soft tissues
PT document use of dynamic or static splints
Active or passive motion ?
Its outcome
any contractures , infection ,
improvement in motor function
improvement in sensation
improvement in functions
5. Past history
TT prohylaxis
drug allergy
DM and other medical illnesses
6. Expectation of Patient
B. Examination - Nerve examination mentioned in previous sections
C.Investigations
1. Electrodiagnostic tests
document injuries , location of inslt , severity of injury , recovery pattern , prognosis ,
objective data for impairment documentation , pathology , selection of optimal muscles for
tendon trasnsfers
Nerve Conductions test (NCT ) and Electromyography (EMG )
shows nerve conductivity , axon or myelin involvement and muscles recruitment capability
Nerve Conduction test
immediately after injury - interference of conduction but impossible to determine severity
Proximal and distal to injury - Normal response
Stimulation across injured segment varies
5-10 days - Reduction in amplitude
- altered configuration of evoked potential
- Neuropraxia - conduction distal to lesion is normal .
Injury Pattern Porximal Stimulation Immediately after injuries after 10 days
(conduction distal to lesion) (conduction across lesion ) (across lesion)
Neuropraxia No response Normal response Normal
Axonotemesis No response Normal No response
or neurotemesis
Proximal or central neurological lesions accessed using retrograde techniques (e.g. F-waves , SSEP )
EMGs
10-14 days after neural injury
spontaneuous rest potentials + sharp waves appear in innervated myotomes with axonal injury
14 to 18 days
fibrillation potentials
denervation potential - last until innervated.
3 Weeks
neural sprouting and increase in amplitude.
2 to 6 months
large potentials remain until reinnervation completed.
Normal insertional activity present immediately after insertional activity.
Sharp waves
Amplitude - 100 - 400 microVoltage
Duration 5 - 150ms
Rate ; 2 - 40 Hz
wide body complexes
Fibrillation
Amplitude ; 5 - 1000 microvoltage
Duration ; 0.5 - 2 ms
Rate; 2-3 Hz.
narrow body
onc can check the waves ;
https://www.researchgate.net/figure/Positive-sharp-waves-and-fibrillation-potentials-were-evident-in-all-four-limbs-upon_fig2_26881420
D.Treatment
Physiotherapy - ROM of the joints to prevent contracutre of joints
Splints
Nerve Repair -we will discuss later on different type of Nerve Repair. Upto now be able to examine
them properly from previous topics .
Tendon Transfer - be able to know different donor tendons for different losses in different nerve
injuries more importantly in Upper Extremity. Radial Nerve examination and its losses is thoroughly
testested in examination.
Its equally important for median , ulnar nerves and brachial plexus injury.
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